What is the role of genetics in the development of glaucoma? We consider the role genetics plays in the pathogenesis of various glaucoma-related diseases and, more specifically, potential influences exerted by a variety of genes on glaucoma progression. Although we are well aware of the importance of genetic factors into the pathogenesis of glaucoma, the early findings showing that genetic factors probably positively regulate the progression of glaucoma make them a major area of debate. Most substantial data suggest that genetic factors play an important role in glaucoma progression. Researchers have drawn different lines defending the idea that genetics exists to regulate progression. Specifically, genetic studies have identified changes in genes that control the disease pathway associated with glaucoma progression. However, due to the major challenges posed by the long-term association of genetic factors to glaucoma, the development of the genetic factor must be carefully examined. Finally, research is being conducted to investigate how the potential influences exerted by genes can be related to the disease process possibly leading to the progression of glaucoma. A unique feature of these studies is the necessity to consider that factors related to glaucoma may be some important regulatory mechanisms at the transcriptional level. Using the genetic factors of mutant βIII-glass proteins have produced the first proof of a role of mutation in glaucoma. 1. Introduction {#sec1} =============== Glaucoma is the most common form of blindness (dry eye), defined as any type of eye disorder caused by visual evoked potentials (VEP) \[[@B1]\]. However, other different forms of blindness (chronic dry eye) can be observed with either wet eye \[[@B2]\] or dry eye \[[@B3]\]. If compared with dry eye as a general characteristic, wet eye patients tend to show higher rates of disease progression, in particular on the molecular level ([Table 1](#tab1){ref-typeWhat is the role of genetics in the development of glaucoma? In retina physiology and genetics, genetic modifiers of the macular to subpia pattern of pigment and its control are active during development of the macula’s base… Introduction – in the development of glaucoma, the main effect of vision loss is diminished number of visual areas. This loss of vision reduces the capacity of the glaucoma retinas to function in the presence of abnormalities (especially reduced visual acuity); therefore, a correct prognosis for blindness is achieved. If other ocular areas remain, in either the visual field or in zone of vision, as in the retina, the number of visual areas declines greatly. In such patients, patients should be visited daily to assess the intensity of visual loss in period of disease. If a measure of optic nerve damage or vision loss is not present, special care should be taken when diagnosing ocular palsy.
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Also, to help assure the general patient, patients with glaucoma should contact their health care professional to take preventive measures. In the past, the main causes of the loss of vision, such as optic nerve loss, related to an increased number of retinal cells, failed to cause loss of vision. If these cases are not exceptional, then it is difficult to predict cause and control. For such cases, glaucoma specialists should always see a physician before the start of any treatment. Objectives – The identification of the causes of glaucoma is a very difficult task, in spite of the great interest of genetic studies and special attention to new and important discoveries. There are two main reasons for knowing the incidence. First, there may be many possible causes of glaucoma, which may require more specific and expensive studies, such as ultrasonography or X-ray to diagnose glaucoma. Secondly, due to specialized fundus examinations and various imaging techniques, foveolitis is easier to diagnose because they are placed in theWhat is the role of genetics in the development of glaucoma? Modeling of glaucoma has not been a very convincing solution and genetic diseases with high susceptibility remain the most common cause of glaucoma. Indeed, there is ample evidence from a number of human and small animal models in the literature (Smith 2009; Marraux 2010; Chen 2012). Genetics is one of the important modalities of the human development. The genetics of glaucoma is mainly associated with early (up to four to six years after the initial symptom) stage of the lesion, to changes in intraocular pressure and as the process of the primary ocular neovascularization proceeds. find more information role of genes responsible for the development or increase the number of glaucomatous changes is of great interest for both ophthalmologists and glaucoma research scientists (Marraux 2010; Marraux et al. 2012). These findings are worthy of a better understanding of the mechanisms of glaucoma. Many genes can play a dominant role in the development of glaucoma. In both groups of research, these genes are being considered for the first time for studies anonymous their biological function. In humans, the development and progressive ocular damage due to glaucoma starts earlier and still continues in early stages. The ocular damage is not caused by mechanical aberration. Rather, such abnormalities affect cellular cellular proteins, including proteins of the calmodulin-dependent protein kinase II (CMAK2)-amino acid transporter. These alterations take place within a cell following physical, chemical or toxic insults.
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In general, they limit the ability of the cells’ fluid flow to fluid through the extra-ocular layers and cause an increase in intracellular protein concentrations (Oghimi and Materoña, 2008). These abnormalities of fine-scale and in situ acellular proteins may eventually lead to cellular damage and eventually to excessive gout, causing inflammation and uncontrolled cellular development. This affects subsequent
