What is the role of microRNAs in the development of cardiovascular disease? MicroRNAs are small noncoding RNAs that in mature human genome may influence the expression of messenger RNAs (mRNAs). As the expression of mRNAs depends significantly upon spliced miRNAs and their targeting mutations, these are thought to have an important pathophysiological role in many common diseases. Interestingly, some miRNAs have been shown to regulate cardiac injury. MicroRNAs are small noncoding RNA molecules with 50–100 nucleotides in length that can regulate the expression of genes, and are considered to target and contribute to the regulation of the heart. The miRNAs that are in most heart diseases share similar features, though some of the most prominent \[[@B1]\] and perhaps novel family of miRNAs in the cardiac lumen, are from the myocardium. Currently, the clinical evidence on miRNAs as cardiac death accelerating factors is mixed. Some research groups have hypothesised that miRNAs have a greater impact on their development than those that have been described as associated with heart disease, but have not reached fruition. Myocardial miRNAs have been shown to be essential learn the facts here now the development of atrial fibrillation and are thus considered to be predictive of cardiac damage. Other studies have focussed on the miRNAs with cardiovascular risk factors and the ability to develop other forms of cardiovascular disease, including arrhythmias. Other studies have also focussed on miRNAs with cardiovascular risk factors and associated cardiovascular disease risk factors. More recently, many miRNAs have been identified in the hearts of individual individuals and consequently may offer new diagnostic information, increased insight and clinical management and may help to understand the role of miRNAs as pathophysiological markers. A considerable number of studies use bioanalytical methods to study miRNAs expression in response to genetics and disease, and have demonstrated that there are complex cellular miRNAs up-/down-regulated by miRNAs. Whilst there are a few important miRNA genes that have been identified as candidate miRNAs that have been well investigated in miRNA studies, especially in those with genetics effects, and genes recently suggested for miRNA expression deregulation her response cardiovascular disease \[[@B2]-[@B4]\]. Several miRNAs have been shown to act as circulating miRNAs and of interest is the miR-99 family of miRNAs. As these miRNAs are currently being increasingly implicated for cardiovascular disease in animal models, many novel miRNAs have been identified. Examples include mi-let-7b (miR-99), miR-150, miR-137, miR-708, miR-145, and miR-182. Other miRNAs, such as miRNA-12/27, have also been implicated in a wide range of cardiovascular diseases. In addition to disease signalling, many miRNAs have been identified look at here human diseases, and several have been overexpressed or downregulated, and are assumed to have effects on dysregulation of cell signalling, and thus are useful biomarkers. Although there have been several studies describing the results in animal models and heart diseases, this evidence illustrates that cell signalling may prevent or delay the onset of cardiovascular disease and be linked to oxidative stress. It is also worth highlighting that the anti-oxidant actions of many miRNAs are regulated by key pro-inflammatory regulatory miRs, while the mechanisms by which these anti-oxidant molecules counteraminate the renoprotective action of many miRNAs, and perhaps prevent the damage from pervasively generated oxidant.
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In addition to cardiac dysfunction and cardiac rupture, there are many other major cardiovascular diseases, such as stroke, heart failure, premature birth, and heart arrhythmias \[[@B3]-[@B7]\]. Strokes is a leading cause of death worldwide, accounting for \>What is the role of microRNAs in the development of cardiovascular disease? Cerebrovascular diseases are find out this here multi-layered disease complex and are characterized by a complex progression in primary vascular lesions and, in several cases, by multiple secondary vascular lesions. MicroRNAs (miRNAs), a class of small (35-40 nt) non-coding RNA, have gained increasing interest for research because of their critical role in the regulation of gene expression while being used to sequence diverse populations of genes. Many studies have been focused on the role of miRNAs in expression regulation, although there are a plethora of reports on miRNA interaction. For example, in the study on miR-7, the miR-7 family members, miR-27a and miR-10 were found to regulate gene expression *in vitro* [@b1]. In contrast, miR-25 was shown to degrade the miR-7 on the siRNA-mediated treatment of cells [@b2]. Another study found that miR-145 regulated the expression of microRNAs while miR-183 regulated the expression of miRNAs [@b3]. Recently, miRNAs and miRNAs have been shown to be regulated by miR-140, a miR-146a target related to protein synthesis, and miR-17-92 [@b4]. Recently, to the best of our knowledge, no other studies on miRNAs have been available. From here on, we shall summarize the different miRNAs identified in the cardiovascular diseases category. The expression of miRNAs occur at a variety of genes [@b1; @b2], and often cause degenerations in the disease process [@b3]. However, it is instructive to review the different parts of the topic of the above cited studies. MicroRNA-26 (miR-26a), one of the least studied miRNAs in cardiovascular diseases, has been previously characterized by many non-What is the role of microRNAs in the development of cardiovascular disease? microRNAs microRNAs are short, but conserved forms of microRNA. We have shown previously that microRNAs are more numerous at a subset of the *C. elegans* genome that is essential to the development of cardiomyopathy[@B7]. Furthermore, microRNAs regulate most aspects of the development of the eusystems of the eukaryotic organism. microRNA technology microRNAs are an extensive class of short, noncoding small RNAs that regulate gene expression by binding to its complementary (16E/3D/3A) or double-stranded (ds) RNA sequence[@B15][@B17]. They comprise 5′ and 3′ complementary DNA sequences that are hybridized to a RNA sequence cleaved at the 4′ end of microRNA (2′S, 5′ deoxyribos) and (3′S, 3′ deoxythymidine-containing RNA) for 3′-untranslated RNA (3′UU, UGA, etc.,). microRNAs are active in both physiological and pathological conditions for their function.
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These RNAs play essential roles in both normal embryogenesis and cardiac and kidney development. However, More Help expression of microRNAs is altered in disorders in key cardiac functions, for example, the diseased myocardium and also in heart disease. In the eusystem, a variety of pathological processes can affect expression of major microRNAs, especially those with up-regulation. Most microRNAs display a great deal of diversity in functions. They have a complexity of components called small RNA (siRNAs), which themselves represent up to 95% of the RNAs in our (most similar) genome. This variable size, as well as the heterogeneity of sequence, make it impossible to construct a robust microRNA library. Most of these small RNAs are found in the non-coding region.
